PeptideWiki
Research Onlyp53-MDM2 Chimeric Anticancer Peptide🔬 Experimental

PNC-27

Also known as: PNC27 · p53-penetratin chimera · MDM-2 targeting peptide

A chimeric anticancer peptide fusing a p53 MDM-2 binding domain with a penetratin membrane-translocation region. Selectively forms pores in cancer cell membranes that overexpress HDM-2 (human MDM-2 oncoprotein), causing rapid membranolysis and cancer cell death while sparing normal cells.

What is PNC-27? A chimeric anticancer peptide fusing a p53 MDM-2 binding domain with a penetratin membrane-translocation region. Selectively forms pores in cancer cell membranes that overexpress HDM-2 (human MDM-2 oncoprotein), causing rapid membranolysis and cancer cell death while sparing normal cells.

How does PNC-27 work? PNC-27 fuses two functional domains: a p53 residues 12-26 sequence (which binds MDM-2/HDM-2) with a penetratin cell-penetrating peptide. HDM-2 is overexpressed at cancer cell surface membranes. PNC-27 binds HDM-2, and the penetratin domain inserts into the lipid bilayer forming pores causing membranolysis within minutes — a non-apoptotic cancer cell killing mechanism.

Benefits of PNC-27: Selective cancer cell membranolysis via HDM-2 binding; Spares normal cells that do not overexpress HDM-2; Rapid cell death mechanism; Active against leukemia, melanoma, pancreatic, breast cancer; Non-apoptotic mechanism — may work in chemo-resistant cancers; Synergy potential with standard chemotherapy

PNC-27 dosage: CAUTION: Anticancer research compound exclusively. Not for human self-administration. In vitro and in vivo research use only.

Research status: Research Only

Source: PeptideWiki — https://www.peptide-wiki.net/peptides/pnc-27

22 AAs
MW: 4,031.73 Da
CAS: 1159861-00-3

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Mechanism of Action

PNC-27 fuses two functional domains: a p53 residues 12-26 sequence (which binds MDM-2/HDM-2) with a penetratin cell-penetrating peptide. HDM-2 is overexpressed at cancer cell surface membranes. PNC-27 binds HDM-2, and the penetratin domain inserts into the lipid bilayer forming pores causing membranolysis within minutes — a non-apoptotic cancer cell killing mechanism.

Benefits

  • Selective cancer cell membranolysis via HDM-2 binding
  • Spares normal cells that do not overexpress HDM-2
  • Rapid cell death mechanism
  • Active against leukemia, melanoma, pancreatic, breast cancer
  • Non-apoptotic mechanism — may work in chemo-resistant cancers
  • Synergy potential with standard chemotherapy

Side Effects & Risks

  • Research compound only — NOT for self-administration
  • Potential off-target membranolytic effects at high doses
  • Systemic safety not established in humans
  • Injection site necrosis risk at wrong concentration

Where to Buy PNC-27

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From

$245.00

Best Price$245.00/ 10mg
$24.50/mg
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Storage & Reconstitution Guide

Storage Temperature

-20°C (lyophilized)

24 months (lyophilized), 28 days (reconstituted)

Reconstitution Solvent

Bacteriostatic water (BAC water)

Swirl gently — do not shake or vortex

Handling Notes

Protect lyophilized peptide from moisture and light. Once reconstituted, keep refrigerated. Discard if solution becomes cloudy or discolored. Use insulin syringe for precise dosing.

Step-by-Step Reconstitution

1

Gather supplies

BAC water, insulin syringe, alcohol swabs, vial

2

Disinfect tops

Swab rubber stoppers of both vials with alcohol

3

Draw BAC water

Pull desired mL of BAC water into syringe

4

Inject slowly

Inject BAC water down the side of the peptide vial, swirl gently to dissolve

Preclinical Research & Reviews1

🧪 In VitroPreclinical

PNC-27 kills cancer cells by selective membranolysis via HDM-2

Michl J, et al.·Oncogene·2006

PNC-27 caused selective membranolysis and rapid death in HDM-2 overexpressing cancer cells while completely sparing normal cells. Active against pancreatic, leukemia, melanoma, and breast cancer lines within minutes.

Speed

Membranolysis within minutes

Mechanism

Pore formation via penetratin domain after HDM-2 binding

Selectivity

100% selective for HDM-2 overexpressing cancer cells

Cancers Tested

Pancreatic, leukemia, melanoma, breast cancer cell lines

Research Protocol

Frequency
Research protocol only
Cycle
Research protocol only
Routes
subcutaneous injectionintravenous injection
Notes
CAUTION: Anticancer research compound exclusively. Not for human self-administration. In vitro and in vivo research use only.
Open Dosage Calculator →

Legal & Regulatory Status

Research StatusResearch Only
WADA Status Not Listed
FDA ClassificationNot Approved
Originsynthetic

Sold for research purposes only. Not for human use. Laws vary by country.

Commonly Stacked With

View all peptide stacks →

External Resources

Chemical Properties

CAS Number
1159861-00-3
Mol. Weight
4,031.73 Da
Formula
C188H293N53O44S
PW

PeptideWiki Research Team

Evidence-based research data sourced from PubMed and ClinicalTrials.gov · Last updated: February 25, 2026

Cite: PeptideWiki. “PNC-27.” peptide-wiki.net/peptides/pnc-27. Accessed 2026.

Frequently Asked Questions About PNC-27

What is PNC-27?

A chimeric anticancer peptide fusing a p53 MDM-2 binding domain with a penetratin membrane-translocation region. Selectively forms pores in cancer cell membranes that overexpress HDM-2 (human MDM-2 oncoprotein), causing rapid membranolysis and cancer cell death while sparing normal cells.

What are the benefits of PNC-27?

Selective cancer cell membranolysis via HDM-2 binding

What are the benefits of PNC-27?

Spares normal cells that do not overexpress HDM-2

What are the side effects of PNC-27?

Research compound only — NOT for self-administration

What is the recommended dosage for PNC-27?

CAUTION: Anticancer research compound exclusively. Not for human self-administration. In vitro and in vivo research use only.

How long should a PNC-27 cycle last?

Research protocol only

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